Is there a disease that mimics gout?
Pseudogout is also called calcium pyrophosphate deposition disease (CPPD). As its name suggests, pseudogout is often mistaken for gout. CPPD typically happens in the knee and other larger joints, including: hip.
How can gout be prevented?
Cut out or limit high-purine foods like red meat and seafood, including shrimp, lobster, mussels, anchovies, and sardines. Limit or eliminate alcohol to prevent another attack. Add dairy products like skim milk and low-fat yogurt, which may protect against gout.
How do you get rid of uric acid crystals?
In this article, learn about eight natural ways to lower uric acid levels.
- Limit purine-rich foods.
- Eat more low-purine foods.
- Avoid drugs that raise uric acid levels.
- Maintain a healthy body weight.
- Avoid alcohol and sugary drinks.
- Drink coffee.
- Try a vitamin C supplement.
- Eat cherries.
What kind of metabolic disorder is Lowe syndrome?
Background. Lowe syndrome, also called oculocerebrorenal syndrome (OCRS) and oculocerebrorenal syndrome of Lowe (OCRL), is an X-linked recessive metabolic disorder described by Lowe and coworkers in 1952. [1] It is a multisystem disorder that primarily affects the eyes, nervous system, and kidneys.
Who is the author of the Lowe syndrome?
Author: Deborah M Alcorn, MD; Chief Editor: Andrew G Lee, MD more… Lowe syndrome, also called oculocerebrorenal syndrome (OCRS) and oculocerebrorenal syndrome of Lowe (OCRL), is an X-linked recessive metabolic disorder described by Lowe and coworkers in 1952. [ 1]
What are the side effects of Lowe syndrome?
Mortality/Morbidity. These patients are predisposed to failure to thrive, severe metabolic acidosis, electrolyte imbalances, and dehydration. Patients with Lowe syndrome also have a tendency to develop pneumonia due to hypotonia and poor cough reflex. Other causes of death include infection and status epilepticus.
How is the diagnosis of Lowe syndrome confirmed?
The diagnosis may be confirmed by testing for reduced levels of the OCRL enzyme in cultured skin fibroblasts or by a DNA test using a blood sample. As some DNA tests may not identify a mutation an enzyme test is the only conclusive test.